CMC – Cardiac Medicine Certification
CMC – Cardiac Medicine Certification is a trademark of its owner, which is not affiliated with and does not endorse Test Treasure Publication or TTP Prep.
CMC – Cardiac Medicine Certification is a trademark of its owner, which is not affiliated with and does not endorse Test Treasure Publication or TTP Prep.
10 sample questions for the CMC – Cardiac Medicine Certification, with the correct answer and an explanation for each. No account needed. Try each one before you open the answer.
Sections covered on the exam: Non-Cardiovascular Conditions, Therapeutic Interventions, Monitoring and Diagnostics.
Question 1 · Non-Cardiovascular Conditions
Answer: D. Conservative medical therapy with several days of inpatient monitoring
In a stable patient with spontaneous coronary artery dissection and preserved flow, conservative management is preferred because most dissections heal on their own within weeks, whereas PCI has high rates of technical failure and can propagate the dissection; several days of inpatient monitoring are advised because early extension can occur. Fibrinolysis can enlarge the intramural hematoma and is avoided. Bypass surgery is reserved for left main or extensive dissection with ongoing ischemia when PCI is not feasible.
Question 2 · Non-Cardiovascular Conditions
Answer: D. Notify the provider for emergent revascularization and anticipate IV heparin
Absent Doppler signals with sensory loss and early motor weakness indicate an immediately threatened limb from acute arterial embolism, requiring an IV heparin bolus and emergent revascularization to prevent irreversible ischemia. Elevation reduces arterial inflow and heat increases metabolic demand and burn risk in an insensate limb. Venous studies and outpatient testing delay definitive treatment.
Question 3 · Non-Cardiovascular Conditions
Answer: B. Metoprolol 5 mg IV every 5 minutes for 3 doses
In acute cocaine intoxication, IV beta-blockers are avoided because they may leave alpha-adrenergic stimulation unopposed, worsening coronary vasospasm and hypertension. Benzodiazepines are first-line to reduce central sympathetic outflow, nitroglycerin relieves vasospasm and ischemic pain, and serial troponin and ECG monitoring are needed to evaluate for myocardial infarction.
Question 4 · Therapeutic Interventions
Answer: A. Raise MAP to ≥ 90 mm Hg and drain CSF to lower spinal fluid pressure to ≤ 10 mm Hg
New lower-extremity weakness after TEVAR indicates spinal cord ischemia from loss of intercostal perfusion; spinal cord perfusion pressure (MAP minus CSF pressure) is improved by raising MAP and draining CSF. Lowering MAP or clamping the drain worsens cord perfusion. Flat positioning with infrequent checks misses a time-sensitive window for reversing the deficit.
Question 5 · Therapeutic Interventions
| Parameter | Value |
|---|---|
| BP | 78/50 mm Hg |
| HR | 112/min, sinus |
| Peak LVOT gradient | 70 mm Hg |
| LVEF | 75%, hyperdynamic |
Answer: B. An IV fluid bolus and phenylephrine, avoiding inotropes
The findings describe dynamic left ventricular outflow tract obstruction, a recognized complication of transcatheter mitral valve replacement. Raising preload with fluid and afterload with a pure vasoconstrictor such as phenylephrine, often with a short-acting beta-blocker to slow the heart and lengthen filling, widens the outflow tract and lowers the gradient. Inotropes, vasodilators and diuretics all make the outflow tract smaller and worsen the obstruction; persistent obstruction may need a procedural correction.
Question 6 · Non-Cardiovascular Conditions
| Sample site | O₂ saturation |
|---|---|
| Right atrium | 64% |
| Right ventricle | 82% |
| Pulmonary artery | 83% |
| Systemic artery | 93% |
Answer: C. Ventricular septal rupture with a left-to-right shunt
An oxygen saturation step-up of more than 5–7% between the right atrium and right ventricle shows oxygenated blood entering the RV through a ruptured septum, which matches the harsh murmur and thrill at the left sternal border. Papillary muscle rupture produces an apical murmur and giant v waves without an RV oxygen step-up. RV infarction and free wall rupture do not create an intracardiac shunt.
Question 7 · Non-Cardiovascular Conditions
Answer: D. Massive leg swelling, bluish skin and weak pedal pulses
Massive swelling with cyanosis and diminishing arterial pulses indicates phlegmasia cerulea dolens, in which venous outflow obstruction raises compartment pressures enough to impair arterial inflow and threatens the limb, requiring urgent catheter-directed or surgical thrombus removal. Asymmetric calf size, low-grade fever and tenderness are expected findings with DVT, and minor injection-site bruising is common with low-molecular-weight heparin.
Question 8 · Non-Cardiovascular Conditions
Answer: D. Obtain serial ECGs and high-sensitivity troponins per protocol
Nitroglycerin relaxes esophageal smooth muscle as well as coronary arteries, so pain relief does not distinguish esophageal spasm from ischemia; a patient with diabetes and cardiac risk needs ACS excluded with serial ECGs and high-sensitivity troponins. A single normal ECG does not rule out ACS. Response to a GI cocktail is equally nondiagnostic, and outpatient esophageal testing is appropriate only after a cardiac cause has been excluded.
Question 9 · Therapeutic Interventions
| Lab | Baseline | 12 h |
|---|---|---|
| Fibrinogen | 310 mg/dL | 118 mg/dL |
| Hemoglobin | 13.1 g/dL | 12.4 g/dL |
| Platelets | 228 × 10³/µL | 201 × 10³/µL |
| aPTT | 34 s | 62 s |
Answer: C. Stop the thrombolytic and anticoagulant infusions and arrange an emergent head CT
A sudden severe headache with a focal deficit during thrombolysis, along with a sharply falling fibrinogen, indicates possible intracranial hemorrhage; all thrombolytic and anticoagulant infusions are stopped immediately and an emergent head CT is obtained. Reducing the rate or continuing the infusion risks extending the bleed. Treating the headache symptomatically delays recognition of a neurologic emergency.
Question 10 · Therapeutic Interventions
Answer: C. Submerge the end of the chest tube 2–3 cm in sterile water
Placing the open end of the chest tube 2–3 cm under sterile water creates an immediate water seal that lets air escape but not re-enter, until a sterile system is reconnected. Prolonged clamping can cause a tension pneumothorax when an air leak is present. The nurse should not remove the tube, and waiting with an open tube allows air to enter the pleural space.
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