Airway, Respiration and Ventilation · 5 min read
Respiratory Assessment, Pulse Oximetry and Waveform Capnography
This chapter covers how to recognize respiratory distress and failure, how to interpret lung sounds, and the strengths and limits of pulse oximetry and waveform capnography. The exam often hands you these findings in words and asks which one matters most.
Work of Breathing and Signs of Failure
Respiratory distress means increased work of breathing with adequate gas exchange: tachypnea, accessory muscle use, retractions (suprasternal, intercostal, subcostal), nasal flaring, tripod positioning, and speaking in short phrases. Respiratory failure means the effort is no longer enough: altered mental status (anxiety, then confusion, then somnolence), cyanosis, falling SpO2 despite oxygen, rising ETCO2, bradypnea, shallow breaths, and, in children, head bobbing and see-saw (paradoxical) breathing. Respiratory arrest is the absence of effective breathing.
Exam Tip: The single most important sign of impending respiratory failure is a change in mental status. A wheezing asthmatic who stops fighting and becomes quiet and drowsy is not improving; they are tiring out. The best answer is to begin assisting ventilations.
Abnormal Breath Sounds
Abnormal Breath Sounds
| Sound | Description | Usual Cause |
|---|---|---|
| Stridor | High-pitched, mostly inspiratory, often audible without stethoscope | Upper airway narrowing: croup, epiglottitis, foreign body, anaphylaxis, burns |
| Wheezes | Musical, mostly expiratory | Narrowed lower airways: asthma, COPD, anaphylaxis, sometimes heart failure |
| Crackles (rales) | Fine popping, often at the bases | Fluid or alveolar reopening: pulmonary edema, pneumonia |
| Rhonchi | Coarse, low-pitched rattling that may clear with cough | Secretions in larger airways: bronchitis, pneumonia |
| Pleural friction rub | Grating, like leather rubbing | Inflamed pleura: pleurisy, pneumonia, PE |
| Diminished or absent | Little or no air movement | Pneumothorax, hemothorax, severe bronchospasm (silent chest) |
Common Trap: Wheezing in an older patient with orthopnea, jugular vein distention, edema and crackles may be "cardiac asthma" from pulmonary edema. A history of heart failure and hypertension steers you to CPAP and nitroglycerin rather than to bronchodilators alone.
Pulse Oximetry and Its Limits
Pulse oximetry estimates the percentage of hemoglobin saturated with oxygen. Target SpO2 is generally 94-99% for most patients, 88-92% for known chronic CO2 retainers, 92-98% after return of spontaneous circulation, and 94% (oxygen only when SpO2 is under 90%) in acute coronary syndrome.
Pulse Oximetry Pitfalls
| Condition | Effect on SpO2 | Why |
|---|---|---|
| Carbon monoxide poisoning | Falsely normal or high | Carboxyhemoglobin reads like oxyhemoglobin; use SpCO |
| Methemoglobinemia | Trends toward about 85% regardless of true value | Abnormal light absorption |
| Poor perfusion, hypothermia, vasoconstriction | Weak or absent signal, unreliable | Probe needs pulsatile flow |
| Nail polish, artificial nails, motion | Inaccurate readings | Light path interference |
| Severe anemia | May read normal | Saturation is normal, but oxygen content is low |
| Lag time | Delayed by 30 seconds or more | Finger probe reflects past events |
Pulse oximetry tells you nothing about ventilation and lags behind real events. Treat the patient, not the number: a cyanotic, gasping patient with an SpO2 of 96% on a cold finger needs airway support.
Waveform Capnography
Capnography measures exhaled CO2. Normal ETCO2 is 35-45 mmHg. A normal capnogram has a square-box shape: phase I (baseline, dead space gas with zero CO2), phase II (a sharp upstroke as alveolar gas arrives), phase III (the alveolar plateau, ending at the ETCO2 value), and phase 0 (a sharp downstroke as inspiration begins).
Capnography Waveform Patterns
| Waveform in Words | Meaning | Paramedic Response |
|---|---|---|
| Square box, 35-45 mmHg | Normal ventilation and perfusion | Continue |
| Shark fin (sloped upstroke, no flat plateau) | Bronchospasm or obstructed exhalation | Bronchodilators; lengthen expiratory time |
| Sudden loss of waveform to zero | Tube dislodged, disconnected, obstructed or esophageal; or arrest | Check tube, circuit, patient immediately (DOPE) |
| Gradual rise over several breaths | Hypoventilation, rising metabolism, fever, bicarbonate | Increase effective ventilation; consider cause |
| Gradual fall | Hyperventilation, falling cardiac output, hypothermia, PE | Slow ventilation or assess perfusion |
| Curare cleft (notch in the plateau) | Spontaneous effort against the ventilator; paralytic wearing off | Reassess sedation and paralysis plan |
| Rising baseline (does not return to zero) | Rebreathing CO2 | Check exhalation valve, circuit |
| Low values during CPR (under 10 mmHg) | Poor compressions or poor prognosis | Improve compressions; consider rescuer change |
| Abrupt rise during CPR to 35-40 mmHg or more | Probable return of spontaneous circulation | Pulse check at the next opportunity |
Waveform capnography is required to confirm endotracheal tube placement and to monitor it continuously. A persistent waveform over six or more breaths confirms tracheal placement better than any other single field method. Lung sounds, chest rise, tube fogging and colorimetric devices are supporting signs only.
Exam Tip: If a question describes a sudden drop of the waveform to a flat line in an intubated patient who still has a pulse, the most likely cause is tube displacement or disconnection. If the ETCO2 falls gradually during a long transport while the patient remains intubated and ventilated, think of falling cardiac output.
Field Scenario: At 0340 you are called to a 34-year-old found unresponsive in a bathroom with a syringe nearby. Respirations are 4/min and shallow, SpO2 is 89% on room air, and nasal capnography shows a tall, slow waveform at 72 mmHg. The paramedic should open the airway, ventilate with a BVM and oxygen at 10-12 breaths/min, then titrate naloxone to adequate respirations rather than to full consciousness. The high ETCO2 confirms hypoventilation; a falling ETCO2 toward the 40s with each breath confirms that ventilation is working.
Pediatric and Geriatric Assessment Points
In children, use the pediatric assessment triangle (appearance, work of breathing, circulation to skin) from the doorway. Grunting is an attempt to create PEEP and signals significant lower airway disease. Bradycardia in a hypoxic child is a pre-arrest sign. In older adults, confusion may be the only sign of hypoxia or hypercapnia.
Key Takeaways
Altered mental status is the key sign that distress has become respiratory failure.
Pulse oximetry misses carbon monoxide, reads near 85% in methemoglobinemia, and lags behind real events.
Normal ETCO2 is 35-45 mmHg; shark fin means bronchospasm, sudden loss means displacement or disconnection.
Waveform capnography is the standard to confirm and continuously monitor tube placement.
Grunting, head bobbing and bradycardia in a child are ominous.
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